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Author Aliev, Gjumrakch.

Title Role of oxidative stress, mitochondria failure, and cellular hypoperfusion in the context of Alzheimer disease : past, present and future / Gjumrakch Aliev.

Publication Info. New York : Nova Science Publishers, Inc, [2013]

Copies

Location Call No. OPAC Message Status
 Axe EBSCO Clinical Nursing E-Book  Electronic Book    ---  Available
Description 1 online resource
text txt rdacontent
computer c rdamedia
online resource cr rdacarrier
Series Neurology--laboratory and clinical research developments
Aging issues, health and financial alternatives
Neurology--laboratory and clinical research development series.
Aging issues, health and financial alternatives series.
Bibliography Includes bibliographical references and index.
Note Print version record.
Contents THE ROLE OF OXIDATIVE STRESS, MITOCHONDRIA FAILURE, AND CELLULAR HYPOPERFUSION IN THE CONTEXT OF ALZHEIMER DISEASE ; THE ROLE OF OXIDATIVE STRESS, MITOCHONDRIA FAILURE, AND CELLULAR HYPOPERFUSION IN THE CONTEXT OF ALZHEIMER DISEASE ; Dedication and Acknowledgments ; Contents ; Preface ; Are Mitochondrial Failures a Key Component in Alzheimer Disease? ; Abstract ; Acknowledgments ; References ; In Vivo and In Vitro Assessment of the Brain Mitochondrial Bioenergetics in Aging Rats ; Abstract ; Introduction ; Materials and Methods ; Animals ; Methods.
In vivo 31P Magnetic Resonance Spectroscopy In Vitro Mitochondrial Respiration ; In Vitro Mitochondrial Oxidative Phosphorylation (Table 2 and Figure 2) ; Endogenous Antioxidants ; Statistical Methods ; Results ; In Vivo 31P Magnetic Resonance Spectroscopy ; Endogenous Antioxidants (Table 3 and Figure 3) ; Discussion ; Acknowledgments ; References ; The Pathophysiology of Cerebrovascular Lesions in Alzheimer Disease ; Abstract ; The Effect of Oxidative Stress on Brain Microvessel Function in AD ; Neuropathologic Features of Cerebrovascular Lesions.
Hypoperfusion as a Factor in the Development of AD Relationships between Apolipoprotein E (ApoE) Genotype, Hypercholesterolemia, and Vascular Changes in AD ; Hypoxia/Ischemia/Reperfusion as Cofactors for Oxidative Stress Induced Cerebrovascular Lesions and their Relationship to AD ; The Role of Mitochondrial Abnormalities in the Patho-Genesis of Oxidative Stress-Induced Brain Lesions in AD ; The Role of Endothelial Vasoactive Substancesin the Pathophysiology of Brain Ischemia/Reperfusion and AD ; Transgenic Animals as Models for Studying Cerebrovascular Lesions in AD.
Prevention of AD Using Selective Pharmacological Treatments Conclusion and Considerations for the Future; Acknowledgments ; References ; Vascular Pathology in the Pathogenesis of Alzheimer Disease ; I. Epidemiological Findings ; II. Elements of Brain Tissue Metabolism; III. Causes and Risk Factors ; Inflammatory System ; Vascular Pathology ; 1. Mitochondrial Dysfunction in AD ; 2. Hypoxia and AD ; 3. History of Stroke, Cerebral Ischemia and AD ; 4. Hypertension and Alzheimer Disease ; Acknowledgments ; References.
Oxidative Stress Induced Mitochondrial Failure and Cellular Hypoperfusion as Primary Pathogenic Factors for the Development and Progression of Alzheimer Disease Abstract ; Introduction ; Cerebral Vascular Energy Requirements: Hypoperfusion as a Key Factor for the Development of AD ; The Effect of Oxidative Stress on Brain Microvessel Function and AŻ Deposition in AD; Morphometric and Neuropathological Features of CerebroVascular Lesions and AŻ in AD ; Relationships between ApoE Genotype, Hypercholesterolemia, AŻ and Vascular Changes in AD.
Subject Alzheimer's disease.
Oxidative stress.
Mitochondrial pathology.
Cell respiration.
Alzheimer Disease
Oxidative Stress
Maladie d'Alzheimer.
Stress oxydatif.
Maladies mitochondriales.
Cellules -- Respiration.
HEALTH & FITNESS -- Diseases -- Alzheimer's & Dementia.
MEDICAL -- Neurology.
Alzheimer's disease. (OCoLC)fst00806532
Cell respiration. (OCoLC)fst00850233
Mitochondrial pathology. (OCoLC)fst01024022
Oxidative stress. (OCoLC)fst01049566
Genre/Form Electronic books.
Other Form: Print version: Role of oxidative stress, mitochondria failure, and cellular hypoperfusion in the context of Alzheimer disease. New York : Nova Science Publishers, Inc, 2013 9781619428782 (DLC) 2011946292
ISBN 9781619428973 (ebook)
1619428970
1619428784 (hardcover)
9781619428782 (hardcover)
Standard No. AU@ 000062325323
NZ1 15023166

 
    
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