| Description |
1 online resource. |
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text txt rdacontent |
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computer c rdamedia |
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online resource cr rdacarrier |
| Series |
Cell biology research progress |
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Public health in the 21st century |
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Cell biology research progress.
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Public health in the 21st century series.
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| Bibliography |
Includes bibliographical references and index. |
| Note |
Description based on print version record and provided by publisher. |
| Contents |
4.5. Nitric Oxide Synthase4.6. Mieloperoxidase; 4.7. Cytochrome P450; 5. In Vivo Effects of Free Radicals; 5.1. Physiological Functions; 5.2. Pathophysiological Effects; Oxidative Damage; 6. Antioxidant Defense Systems; and Oxidative Stress; 7. Biomarkers of Oxidative Stress; 8. Oxidative Stress and Diseases; 8.1. Cardiovascular Diseases; 8.2. Hypertension; 8.3. Atherosclerosis; 8.4. Metabolic Syndrome and Diabetes; 8.5. Neurodegenerative Diseases; 8.6. Cancer; 8.7. Rheumatoid Arthritis; 8.8. Aging; Conclusion; References; Chapter II: Pathophysiology of Sepsis and Septic Shock; Abstract. |
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Abbreviations2. Introduction; 3. Pathophysiology of Sepsis; 3.1. Basic Molecular Mechanisms of Inflammation in Sepsis; 3.1.1. Pathogen-Host Interaction; 3.1.2. Neutrophils; 3.1.3. Cytokines; 3.1.4. Coagulation Disorders in Sepsis; 3.1.5. Endothelial Cells; 3.1.6. Complement Activation; 3.2. Immunoparalysis; 4. Pathophysiology of Septic Shock; Conclusions and Future Goals; References; Chapter III: Inflammation; Abstract; Abbreviations; 2. Introduction; 3. Current Concepts of the Inflammatory Response; 3.1. Cellular Mediators; 3.1.1. Neutrophils; 3.1.2. Macrophages; 3.1.3. Other Cells. |
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3.2. Cytokines, Chemokines and Lipid Mediators3.2.1. Lipid Mediators; 3.2.1.1. Resolvins Improve the Course of Inflammation; 3.2.2. Novel Cytokines; 3.3. The Role of Toll-Like Receptors; 3.3.1. Clinical Aspects; 3.4.1. Nuclear Factor kappaB; 3.4.1.1 Nuclear Factor kappaB and Oxidative Stress; 3.4.1.2. Nuclear Factor kappaB and SIRS/Sepsis; 3.4.2. STAT-3; 4. The Relationship between Ros Production; and the Inflammatory Response; in Critical Patients; 4.1. Gene Expression; 4.2. Paracrine Effects of ROS; 5. Biomarkers of the Inflammatory Response; in Critically Ill Patients. |
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5.1. Clinical Trials that Support the Prognostic Values of Procalcitonin5.2. Others Proposed Biomarkers in Critical Ill Patients; Conclusions and Perspectives; References; Chapter IV: Mitochondrial Dysfunction; Abstract; Abbreviations; 1. Introduction; 2. Mitochondrial Physiology; and Pathophysiology; Oxidative Phosphorylation; Sources of Reactive Species in Mitochondria; Mitochondria as an Oxygen Sensor; Role of Mitochondria in Apoptosis; 3. Mitochondrial Dysfunction; 3.1. Evidence Supporting the Occurrence of Mitochondrial Dysfunction; in Sepsis. |
| Subject |
Oxidative stress.
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Critically ill.
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Oxidative Stress |
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Stress oxydatif.
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Malades dans un état critique.
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MEDICAL -- Terminal Care.
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Critically ill
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Oxidative stress
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| Added Author |
Rodrigo, Ramon.
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Dessauer, Bettina von.
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| Other Form: |
Print version: Oxidative stress and the critically ill patient New York : Nova Biomedical, c2012. 9781620815762 (hardcover) (DLC) 2012011767 |
| ISBN |
9781620815830 (ebook) |
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9781620815762 (hardcover) |
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1620815761 |
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1620815834 (electronic bk.) |
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9781620815830 (electronic bk.) |
| Standard No. |
AU@ 000056863815 |
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