| Description |
1 online resource (xxi, 314 pages) |
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text txt rdacontent |
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computer c rdamedia |
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online resource cr rdacarrier |
| Bibliography |
Includes bibliographical references and index. |
| Contents |
Lesions associated with the classic triad of Parkinsonian motor features / Stanley Fahn and Glenda M. Halliday -- Lesions associated with motor speech / Anna Rita Bentivoglio, Davide Quaranta, and Aileen K. Ho -- Lesions associated with eye movements / Andrew W. Michell, Roger A. Barker, and Glenda M. Halliday -- Olfactory dysfunction in Parkinson's disease and related disorders / Richard L. Doty, Christopher Hawkes, and Henk W. Berendse -- Lesions associated with autonomic dysfunction. Swallowing disorders and drooling / Maria G. Cersosimo and Eduardo E. Bennarroch. Gastrointestinal disorders / Adolfo Mínguez-Castellanos and Dominic B. Rowe. Orthostatic hypotension / Spiridon Papapetropoulos and Kathryn K. Post -- Lesions associated with pain and sensory abnormalities / Andreas Hartmann and Glenda M. Halliday -- Lesions associated with sleep disturbances / Marcus M. Unger [and others] -- Lesions associated with depression and apathy / Uwe Ehrt, Kenn F. Pedersen, and Dag Aarsland -- Lesions associated with dyskinesias and the dopamine dysregulation syndrome / Andrew H. Evans -- Lesions associated with visual hallucinations and psychoses / David R. Williams and Werner Poewe -- Lesions associated with cognitive impairment and dementia / Jonathan Evans, Tamas Revesz, and Roger A. Barker -- Systemic manifestations of Parkinson's disease / Dominic B. Rowe. |
| Summary |
Parkinson's disease becomes apparent only after substantial loss (& 60%) of the dopamine neurons in the substantia nigra. By this time there has already been widespread neural inclusion formation in the peripheral and central nervous system of patients with the disease, although this has only been recognized more recently. Degeneration in these widespread regions of the peripheral and central nervous system is now known to impact on disease symptoms, progression and treatment over time. This book aims to provide a comprehensive review of these non-dopamine lesions in Parkinson's disease by assessing our current knowledge of their presence and pathophysiology, how they relate to different symptoms and, where relevant, discuss how they may be potentially treated. |
| Note |
Print version record. |
| Language |
English. |
| Subject |
Parkinson's disease -- Pathophysiology.
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Dopamine.
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Parkinson Disease -- physiopathology |
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Brain -- pathology |
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Dopamine |
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Parkinson Disease -- complications |
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Maladie de Parkinson -- Physiopathologie.
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Dopamine.
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HEALTH & FITNESS -- Diseases -- Alzheimer's & Dementia.
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MEDICAL -- Neurology.
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Dopamine
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Parkinson's disease -- Pathophysiology
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| Added Author |
Halliday, Glenda M.
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Barker, Roger A., 1961-
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Rowe, Dominic B.
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| Other Form: |
Print version: Non-dopamine lesions in Parkinson's disease. New York : Oxford University Press, 2011 9780195371086 (DLC) 2010006010 (OCoLC)529295607 |
| ISBN |
9780199707881 (electronic bk.) |
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019970788X (electronic bk.) |
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0195371089 (alk. paper) |
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9780195371086 (alk. paper) |
| Standard No. |
DEBBG BV043074583 |
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DEBSZ 424613808 |
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